Kidney Health

Diabetes is currently the most common cause of chronic kidney disease and failure in the United States. Over time, diseased kidneys may lose their ability to filter substances all together, which can result in end stage renal disease. Patients are counseled on dietary carbohydrate and sugar intake to reduce the complications of diabetes. Additionally, sugar consumption and fructose consumption have been widely studied to further investigate the long term health effects on the kidneys.

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Many published animal studies feed animals with excessive fructose, in some cases as much as 60% of total daily calories, to determine possible pathways that may be involved in kidney disease. It is important to note this amount of fructose far exceeds what people normally consume. Additionally, dietary fructose is typically not consumed in an isolated form but rather accompanied by other sugars like glucose.

Dietary fructose consumption does not contribute to poor kidney health outcomes when consumed in normal amounts in the human diet. However, it is important to note that obesity and diabetes can be inter-related in individuals with chronic kidney disease and metabolic syndrome.

Research Study Summaries

Calorie Control Council Response to Johnson et al

The effect of fructose on renal biology and disease

Calorie Control Council Response to Douard et al

Dietary Fructose Inhibits Intestinal Calcium Absorption and Induces Vitamin D Insufficiency in CKD

Calorie Control Council Response to Choi Editorial

The not-so-sweet side of fructose

Fructose, but not dextrose, accelerates the progression of chronic kidney disease.

The authors have adopted the hypothesis of Bray et al that fructose/HFCS are uniquely responsible for the obesity/metabolic syndrome epidemic. They have extended the Bray hypothesis to suggest in this paper that fructose/HFCS are also unique contributors to chronic kidney disease.